Full Article: PDF
Scientific Object Identifier: http://s-o-i.org/1.1/TAS-10-138-14
DOI: https://dx.doi.org/10.15863/TAS.2024.10.138.14
Language: English
Citation: Turgunov, A.M. (2024). Analysis of the mathematical model of regulatorika of the liver cells and hepatitis b viruses. ISJ Theoretical & Applied Science, 10 (138), 144-150. Soi: http://s-o-i.org/1.1/TAS-10-138-14 Doi: https://dx.doi.org/10.15863/TAS.2024.10.138.14 |
Pages: 144-150
Published: 30.10.2024
Abstract: Every year, between 10 and 30 million people worldwide become infected with the hepatitis B virus (HBV). Most of those infected are children and adolescents. The hepatitis B virus uses the cell's resources to begin synthesizing, or replicating, the components needed to build new viruses. DNA polymerase triggers the liver cell to make copies of the hepatitis B virus DNA and thus create more viruses. When the cell finally makes these components, they assemble into a complete virus and these viral copies are released into the blood. After the virus has assembled, so-called waste products remain in the form of surface proteins that are released into the blood. The new hepatitis virus goes on to infect other liver cells and continually repeats this efficient and rapid reproduction process. In fact, thousands of new virus particles can be produced in each liver cell in a single day. The virus easily penetrates the liver, the largest internal organ. If the hepatitis virus manages to evade the body's immune system and encounters a liver cell (hepatocyte), the outer shell of the virus attaches to the surface of the liver cell and the genetic material from the core of the virus is incorporated into the hepatocyte.
Key words: hepatitis B virus, hepatocyte, molecular-genetic systems, regulatorika, mathematical model, sequential integration.
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